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One of the mecha-nisms involved in disrupting this interaction is the activation of p53 by the ataxia-telengiectasia mutated (ATM) and ataxia-telengiectasia and Rad3-related (ATR)protein kinases which are the major DNA damage sensors mediating the rapiddegradation of MDM2 and MDM4 . This is referred toas the H band, which is occupied by thick filaments and is devoid ofthin filaments.
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